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| Naji, M. |
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| Motta, Antonella |
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| Aletan, Dirar |
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| Mohamed, Tarek |
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| Ertürk, Emre |
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| Taccardi, Nicola |
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| Kononenko, Denys |
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| Petrov, R. H. | Madrid |
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| Alshaaer, Mazen | Brussels |
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| Bih, L. |
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| Casati, R. |
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| Muller, Hermance |
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| Kočí, Jan | Prague |
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| Šuljagić, Marija |
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| Kalteremidou, Kalliopi-Artemi | Brussels |
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| Azam, Siraj |
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| Ospanova, Alyiya |
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| Blanpain, Bart |
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| Ali, M. A. |
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| Popa, V. |
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| Rančić, M. |
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| Ollier, Nadège |
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| Azevedo, Nuno Monteiro |
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| Landes, Michael |
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| Rignanese, Gian-Marco |
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Gustafsson, Anna
Lund University
in Cooperation with on an Cooperation-Score of 37%
Topics
Publications (6/6 displayed)
- 2024Impact of storage time prior to cryopreservation on mechanical properties of aortic homograftscitations
- 2022Crack propagation in articular cartilage under cyclic loading using cohesive finite element modelingcitations
- 2020The influence of microstructure on crack propagation in cortical bone at the mesoscalecitations
- 2019The role of microstructure for crack propagation in cortical bone
- 2019An interface damage model that captures crack propagation at the microscale in cortical bone using XFEMcitations
- 2019Crack propagation in cortical bone is affected by the characteristics of the cement line : a parameter study using an XFEM interface damage modelcitations
Places of action
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thesis
The role of microstructure for crack propagation in cortical bone
Abstract
Healthy cortical bone tissue is both tough and strong and has a unique ability to resist fracture. One reason is the hierarchical structure of the tissue where toughening mechanisms at all length scales act to slow down or stop a propagating crack. The most potent toughening mechanisms arise at the microscale when cracks interact with the osteonal microstructure and deflect along weak interfaces. However, cortical bone is a living material and the tissue properties change over time. With aging the properties are known to deteriorate. Yet, the link between age-related structural and compositional changes and impaired fracture resistance in old bone is not fully known. This is key for understanding and being able to predict the increased risk for fracture with age and in patients with osteoporosis. The aim of this thesis is to understand the role of microstructure for crack propagation in cortical bone. Both experimental and numerical techniques have been used to evaluate the importance of mechanical properties and microstructural distributions for how cracks interact with the microstructure. In the experimental part, in situ loading in combination with digital image correlation and small- or wide-angle x-ray scattering was used to simultaneously measure deformation at meso- and nanoscale in cortical bone. Micro-CT analysis of the bone samples was performed after the tests and showed that the crack trajectory to a large extent followed the microstructure. In the numerical part, the extended finite element method (XFEM) was adopted to explicitly simulate crack propagation in cortical bone at the microscale. The key feature is a new interface damage model in 2D that captures crack deflections at osteon boundaries, as seen in experiments and which previous XFEM models were not able to predict. The modelling framework has been applied to simplified geometries comprising one osteon with different orientations to look at the effect of the microstructural distribution. These models have also been used in a parameter ...